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Osteoarthritis is characterized by the break down of the cartilage that covers the ends of the bones and cushions the joints, disintegration of the underlying bone, and the formation of painful bone spurs or osteophytes around the affected joint.
You do not "catch" osteoarthritis ...
it is a deterioration of cartilage
that covers the ends of the bones.
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Osteoarthritis evolves over time from lifestyle factors (nutrition, activity, trauma). Osteoarthritis rarely develops before the age of forty, but it affects over 80% of those past the age of fifty. However, it may be so mild that a person is unaware of it until it shows up on an x-ray. It typically runs in families, and afflicts almost three times as many women as men. There are approximately 15.8 million incidences of osteoarthritis in the United States.
Some common symptoms of osteoarthritis include stiffness and pain on joint motion. Most often osteoarthritis comes on gradually over a period of years. Inflammation is not usually present at first but in the later stages, inflammation, enlargement of the joint, and muscle contractures may occur. Joint mobility may become limited, and movement may be accompanied by a grating sensation. OUCH -- very painful! This limited mobility and grating occurs when the cartilage breaks down and the normally smooth sliding surfaces of the bones become pitted and irregular. The tendons, ligaments, and muscles holding the joint together become weaker, and the joint itself becomes deformed, painful, and stiff. The affected joints make popping, clicking, and banging noises when they move.
 As osteoarthritis advances fractures become an increasing risk as bone spurs (osteophytes) tend to develop. These spurs, which can be detected by x-ray, develop near degenerated cartilage in the neck or lower back and can cause instability of equilibrium or imbalance. Weight-bearing joints (hands, neck, spine, hips, knees, etc.) are most commonly affected with osteoarthritis and specific symptoms generally vary according to which areas/joints are affected. One particularly interesting trait of osteoarthritis is that there is often a marked lack of correlation between the severity of the joint degeneration (as shown by x-ray) and the amount of pain felt by the afflicted person.
Those at a particularly high risk for osteoarthritis include persons with a prior joint injury/trauma (fractures along joint surfaces, surgery, etc.), a congenital joint deformity or previous joint inflammation from a disease such as gout or rheumatoid arthritis. Obesity also contributes to osteoarthritis as the excess weight puts added stress and wear and tear on the joints. Hormones may also play some part in the development of osteoarthritis. Since women are more likely to suffer from the disease than men estrogen is suspect. Those using growth hormones, which in excessive levels have a negative effect on joints and patients with hypothyroidism also run an increased risk of osteoarthritis.
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[Main Arthritis Page] [OA-Kit]
[Nutrient Associations, Lifestyle Changes, Medical Options and Precautions]
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Osteoarthritis Treatment
"If we examine data collected from the earliest lesions to the most advanced stages of clinical osteoarthritis, the process contributing to osteoarthritis appears to be able to be stopped and, in many cases, reversed. [J.H. Bland and S. M. Cooper,"Osteoarthritis : A Review of the Cell Biology Involved and Evidence for Reversibility: Management Rationally Related to Known Genesis and Pathophysilolgy," Seminars on Arthritis and Rheumatism 14 (1984): Pgs 106-133. The major therapeutic goal appears to be enhancing repair of the collagen matrix and regeneration by the connective-tissue cells."
Several studies have been conducted to determine whether or not the drugs typically used to treat osteoarthritis (nonsteroidal anti-inflammatory drugs and aspirin) are accomplishing the goal of promoting the repair and regeneration of tissue destroyed by osteoarthritis. One of these studies observed what happened over a ten-year period in patients with advanced osteoarthritis of the hip who were given no treatment with the normal drugs. At the end of the study most of the patients showed improvement, including a complete recovery in fourteen of thirty-one hips. Obviously these results and other results raise serious concerns that medical interventions may actually promote disease progression.
"Experimental studies have shown that
NSAID use is associated with acceleration
of osteoarthritis and increased joint destruction"
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Some of the side effects of aspirin and nonsteroidal anti-inflammatory drugs (NASIDs)--such as ibuprofen (Motrin, Nupron, Advil), piroxicam (Feldene), diclofenac (Voltaren),fenoprofen (Nalfon), indomethacin (Indocin), naproxen (Naprosyn),tolmetin (Tolectin), and sulindac (Clinoril)-- are well-known: gastrointestinal upset, ulcer formation, headaches, dizziness, etc. A less well-known side effect is further degeneration of the joint cartilage. Experimental studies have shown that NSAID use is associated with acceleration of osteoarthritis and increased joint destruction [P.M. Brooks, S.R. Potter and W.W. Buchanan,"NSAID and Osteoarthritis-- Help or Hindrance," J Rheutatol 9 (19820: 3-5. N.M.Newman, R.S.M. Ling, "acetabular Bone Destruction Related to Non-Steroidal Anti-inflammatory Drugs," Lancet 2 (1985):11-13] Simply stated, NSAIDS appear to suppress the symptoms but accelerate the progression of osteoarthritis.
So besides these drugs with their somewhat questionable benefits what is available to osteoarthritis sufferers? Our Osteoarthritis Disease Self-management Program will give you nutrient association information as well as other helpful lifestyle changes for managing the disease.
Let me remind you that all our products carry a 30-60 day money back
satisfaction guarantee, so there is no risk in trying a focused nutrition program for 45-60 days.
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Knowledge gives a person many options for managing osteoarthritis (OA) and they can then personally take charge of the effect this disease is having on their life.